Copyright © 2024 AudiologyOnline - All Rights Reserved

AudiologyOnline Phone: 800-753-2160

Exam Preview

Relationships Between Recreational Noise Exposure and Auditory Function, presented in partnership with Seminars in Hearing

View Course Details Please note: exam questions are subject to change.
Print

1.  What lasting physiological effect of noise exposure has been shown by Kujawa and Liberman, even after TTS has recovered, using mice as a model?
  1. Permanent change in behavioral threshold
  2. Chronic tinnitus
  3. Lasting decrease in OAE amplitude
  4. Lasting decrease in ABR amplitude
2.  What is the primary pathology used to explain the lasting effects of noise exposure on the mice who recovered from a TTS, as shown by Kujawa and Liberman?
  1. Outer hair cell loss at/above exposure frequency.
  2. Decreased number of synaptic contacts at/above exposure frequency.
  3. Neural degeneration at/above exposure frequency.
  4. Strial thinning at/above exposure frequency.
3.  The original definition explicitly proposed for hidden hearing loss was:
  1. Decreased DPOAE amplitude as a result of cochlear synaptopthy.
  2. Decreased speech in noise identification scores as a result of cochlear synaptopthy.
  3. Decreased ABR wave I amplitude as a result of cochlear synaptopthy.
  4. Tinnitus as a result of cochlear synaptopthy.
4.  The amount of noise exposure needed to induce synapse loss:
  1. Is the same for all species tested to date.
  2. Is different for different species - it takes more noise exposure to induce synapse loss in a monkey than in a rodent.
  3. Is different for different species - it takes less noise exposure to induce synapse loss in a monkey than in a rodent.
  4. None of the above - the only available data come from rodents.
5.  Cochlear synaptopathy has been found in human temporal bones,
  1. and it has been associated with aging in studies to date.
  2. and it has been associated with noise-exposure in studies to date.
  3. and it has been associated with BOTH aging and noise exposure in studies to date.
  4. None of the above - this pathology has not been found in human temporal bones.
6.  Noise exposure that induces a TTS can result in:
  1. Immediate synapse loss
  2. Decreased ABR amplitude
  3. Long-term spiral ganglion loss
  4. All of the above
7.  In human research investigations recruiting normal hearing populations exposed to recreational noise:
  1. Noise exposure is reliably associated with wave I amplitude deficits across studies.
  2. Noise exposure is reliably associated with wave I amplitude deficits, but only in studies that recruit participants with diabetes.
  3. Noise exposure is not reliably associated with wave I amplitude deficits, although there are some studies that report such relationships.
  4. Noise exposure is not reliably associated with wave I amplitude deficits in any studies to date.
8.  In human research investigations recruiting normal hearing populations exposed to firearm noise:
  1. Noise exposure is reliably associated with wave I amplitude deficits across studies.
  2. Noise exposure is reliably associated with wave I amplitude deficits, but only in studies that recruit participants with diabetes.
  3. Noise exposure is not reliably associated with wave I amplitude deficits, although there are some studies that report such relationships.
  4. Noise exposure is not reliably associated with wave I amplitude deficits in any studies to date.
9.  What were the most common exposures to noise (Le Prell, et al., 2018)?
  1. Bars
  2. Dance Clubs
  3. Music Player use
  4. All of the above
10.  Noise exposures that do not result in permanent threshold shift can cause ABR amplitude _____ and “hidden” hearing loss .
  1. Increase
  2. Decrease
  3. Stability
  4. No change

Our site uses cookies to improve your experience. By using our site, you agree to our Privacy Policy.